The discovery could help clinicians diagnose children who carry mutations in the gene, called SCN2A, and gauge their responses to potential therapies.

The NIH neurologist talks about her research, her family and how mental health labels can be limiting.

The framework, inspired by the polygenic risk score, considers the cumulative effect of neuronal connections.

Mice with microglia missing receptors for the neurotransmitter serotonin since birth have too many synapses and show social difficulties in adulthood.

The gene-editing advances make it easier to target specific tissues in mice and detect off-target effects.

The drugs may reopen a critical window during development in which the brain can more easily adjust its connections.

This month’s newsletter tackles null findings from an attempted replication of a “revolutionary” MRI approach and an analysis of family genetics.

Many genes related to the condition play a role in the internal scaffolding of cells, and cytoskeletal disruptions can affect neurodevelopment and behavior.

A subset of thalamic neurons support an aspect of social behavior called social recognition, according to experiments in mice.

A text-predicting chatbot parses text from conversations in a way that parallels brain-activity patterns associated with speech production and comprehension.