Findings on microglia and other brain cell types bolster the animal’s validity as a model system for the condition.

Our Twitter feeds were awash with research activity this week, including a review of autism intervention studies and resources to build better data-wrangling skills.

The prize recognizes Geschwind’s contributions to our understanding of autism genetics.

Overexpressing genes involved in the pathway, which regulates protein quality, changes repetitive behaviors in the animals.

Cortical organoids that contain non-neuronal brain cells called microglia may reflect human neurodevelopment more faithfully than those that don’t.

Treatments that counteract the effects of an SCN2A mutation in mice increase the animals’ sociability in adulthood, according to a new unpublished study.

Animal models of autism rooted in exposure to maternal antibodies hint at different mechanisms.

Mock viral infections impair social memory in mice with a mutation tied to autism, and autistic boys are more likely than their non-autistic peers to have had serious infections early in life.

An autism-linked mutation could make the brain grow unusually large by prompting cells to express a chemical signal better known for its connection to cancer.

Misaligned gene expression maps suggest that some autism-linked genes play distinct roles in mouse and human brains.