Maternal infection may accelerate expression of autism genes
Exposure to infection in utero may speed up the expression of many genes linked to autism — and hasten changes in brain anatomy.
Exposure to infection in utero may speed up the expression of many genes linked to autism — and hasten changes in brain anatomy.
Marmosets exposed to a mock infection in the womb have altered vocal development and diminished social interest; exposed macaques show changes in brain structure and function.
Researchers have created a microscopic particle that traps immune molecules found in a woman that are linked to autism in her child.
Brain tissues from people with autism of unknown cause and from people with either of two genetic forms of the condition all show similar patterns of methyl groups on DNA.
Scientists are playing catch-up as microbiome-based treatments for autism proliferate.
Exposure to inflammation in the womb may affect the brain and behavior of males and females differently.
A brain region that orchestrates responses to social cues and aids decision-making may be off tempo in autism.
Understanding the interactions between the placenta and the uterine lining could explain how maternal immune activation leads to autism.
A cell atlas and a cell-culture method promise to advance scientists’ understanding of the placenta and its effects on a fetus.
Children who contract serious infections may have an increased likelihood of being diagnosed with autism.