Drug helps mice with autism-linked mutation recognize others
Mice that model 22q11.2 deletion syndrome lack social memory, but that trait can be reversed using a drug that targets the flow of potassium ions in neurons.
Mice that model 22q11.2 deletion syndrome lack social memory, but that trait can be reversed using a drug that targets the flow of potassium ions in neurons.
Autistic people have distinct patterns of brain development, which sometimes result in differences in brain structure. Here’s what we know about those differences.
Doctors and scientists should consider sleep problems an integral part of autism and begin to study them in more rigorous ways.
Dampening overactive brain circuits alleviates social and memory problems in a mouse model of 22q11.2 deletion syndrome.
People with mutations in a gene called TBR1 have unusual features in several brain regions, along with autism traits and developmental delay.
Lattice-like structures that surround neurons may be overly abundant — or scarce — in brain regions of three autism mouse models.
Triggering an immune defense in newborn male mice missing a copy of TSC2, a gene linked to autism, impairs the mice’s social memory.
Applying sound waves to a mouse’s brain provides a safe way to push molecular probes into the brain to manipulate activity there.
Mice lacking one copy of a gene linked to autism are small and show striking changes in the number and quality of their cries.
A new brain implant pumps chemical signals directly into a target region to ease seizure-like activity in mice.